Advances in Immunology, Vol. 47 by Frank J. Dixon (Eds.)
By Frank J. Dixon (Eds.)
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Additional resources for Advances in Immunology, Vol. 47
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Fig. 2), does not exclude the possibility that some of the antigen-specific T cells may form IgE-BFs. Indeed, we have obtained an antigen-specific mouse T cell clone and antigen-specific T cell hybridomas which produce IgE-BFs upon incubation with OVA-pulsed syngeneic macrophages. It was found that some of the antigen-specific T cell hybridomas such as 231F1, constitutively formed GIF, and produced both IgE-SFs and IgG-suppressivefactors upon incubation with OVA-pulsed macrophages (Jardieu et a l .
Indeed, Sarfati et al. (1987b) identified FceRII and its fragments from HTLV-I-transformed cell line cells by sodium dodecyl sulfate-polyacrylamide gel electrophoresis. Immunofluorescence staining of peripheral blood lymphocytes with monoclonal anti-FceRs failed to demonstrate FccRIIs on normal T cells. However, Young et al. (1984) established a human T cell clone bearing FceR from peripheral blood lymphocytes of atopic dermatitis patients. , 1987) responded to homologous IgE and to antibodies reacting to FccRIIs for the formation of IgE-BFs.